Claims

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As of snapshot 27745 under ledger-default@0.3.0.

Claim	Type	Assessment	Score	Review checks	References
In the same skin-infection model, a later neutrophil-recruitment phase depended on Toll-like receptor and CXCL2 signaling, marking a transition to canonical pathogen-driven innate immunity.	CAUSAL	Supported	0.9335	1 of 4	125
In a skin-infection model with pathogenic bacteria and fungi, microbial adhesion initiated an early PRR-independent neutrophil-recruitment phase by engaging Piezo1 and promoting leukotriene B4, interleukin-1α, and CXCL1 signaling.	CAUSAL	Supported	0.9601	1 of 4	118