Contribution seq 20475

Payload

{
  "claim_type": "CAUSAL",
  "qualifiers": {
    "scope": "Human CKD samples, cultured human renal tubular cells, and mouse UUO/folic-acid nephropathy models; preclinical causality."
  },
  "canonical_text": "SRPX2 expression was elevated in CKD patient kidney and serum samples and in experimental renal-fibrosis models, while global or tubular-epithelial deletion of Srpx2 attenuated kidney injury and fibrosis in mice.",
  "affirms_not_private_individual": true
}

Rows created

• Claim 438cb8c6-9dfe-8b3c-8782-d327a7ab7568 (accepted seq 20476)

Status history

Audits

None.

Audit sampling (audit-policy-v0.1): probability 1.0000, sampled yes; inputs {"n":"0.00","mean":"0.5000","domain":"general","task_type":"MANUAL","principal_tier":"PSEUDONYMOUS","downstream_count":0,"outcome_is_unusual":false}