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Ontogeny and transcriptional regulation of Thetis cells PRIMARY_TEXT

Nature · no page hash: the reader saw a rendering, not the bytes · created seq 21626

Link: https://www.nature.com/articles/s41586-026-10198-z · read at 2026-09-29T02:07:55Z · held by reference: only the link, the quoted excerpts with their hashes, and the page hash when the reader had the bytes are stored

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FETCHED at 2026-09-29T04:30:06Z · https://www.nature.com/articles/s41586-026-10198-z?error=cookies_not_supported&code=61615a95-ce61-4705-b9e8-80021a410e56 · seq 25136

Server hash sha256:f03d699053c3e807dee5950c129a24faee3a172a3e4dca150cada0331faf7a63 · 674576 bytes · text/html. The reader's record is unchanged; this is the server's finding beside it.

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Ablation of RANKL expression in LTo cells using Twist2 cre Tnfsf11 fl/fl mice led to an almost complete absence of TC I.not found — this server looked at the page and did not find the passage
Despite transcriptional similarity to myeloid-derived conventional dendritic cells, we show that TCs descend from the common lymphoid progenitor.verbatim — the passage is on the page, character for character
Ablation of PU.1 in RORγt+ cells with Rorgt cre Spi1 fl/fl mice resulted in an almost complete absence of TC I–IV in P14 mLN, but no impairment in LTi cells.not found — this server looked at the page and did not find the passage

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QUOTE {}Ablation of RANKL expression in LTo cells using Twist2 cre Tnfsf11 fl/fl mice led to an almost complete absence of TC I.
QUOTE {}Despite transcriptional similarity to myeloid-derived conventional dendritic cells, we show that TCs descend from the common lymphoid progenitor.
QUOTE {}Ablation of PU.1 in RORγt+ cells with Rorgt cre Spi1 fl/fl mice resulted in an almost complete absence of TC I–IV in P14 mLN, but no impairment in LTi cells.

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