Claim

Model ledger-default@0.1.0 ledger-strict@0.1.0 ledger-default@0.2.0 ledger-strict@0.2.0 ledger-default@0.3.0 (default) ledger-strict@0.3.0 Snapshot seq

Topics

Biology · 1efd24fe

Assessment

Why?

Strongest counted support: The supplied passage directly reports mutant-analysis evidence implicating MCA1 mechanosensitive channels in long-distance wound communication. weight 1.800000 evidence

Suppressed as dependent: The primary study reports neighboring-root depolarizations within milliseconds and implicates MCA1 channels in long-distance wound communication.

Review gaps: qualifiers reviewed, opposing search done, independence reviewed

What would most change this: One independent, direct measurement that contradicts this claim would move it from Leans supported to Leans contradicted.

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Checked as part of

Statements this claim was broken out of. The claim is scored on its own; a statement's reading only counts its claims.

• “In Arabidopsis seedlings, wounding one lateral root triggers rapid electrical and calcium signals in neighboring lateral roots, with MCA1 channels...” · 2 claims · 29 September 2026

Evidence for

• The primary study reports neighboring-root depolarizations within milliseconds and implicates MCA1 channels in long-distance wound communication.

  From MCA1 mechanosensitive channels enable fast communication of wound signals between lateral roots Read 2026-09-29 Relevance STRONG Steps 0 Counted

  The abstract reports mutant-analysis evidence implicating MCA1 in long-distance communication.

• The supplied passage directly reports mutant-analysis evidence implicating MCA1 mechanosensitive channels in long-distance wound communication.

  From MCA1 mechanosensitive channels enable fast communication of wound signals between lateral roots Read 2026-09-29 Relevance DIRECT Steps 0 Counted

  The excerpt explicitly states MCA1's role in mediating the communication.

Evidence against

None.

Qualifications

• The passage limits the result to injury-evoked electrical and calcium responses in neighboring lateral roots and says mutant analyses highlight MCA1 roles; it does not state sufficiency or a broader systemic range.

  From MCA1 mechanosensitive channels enable fast communication of wound signals between lateral roots Read 2026-09-29 Relevance DIRECT Steps 0 Counted

  The target should be read as a preclinical mechanistic contribution under the reported injury and neighboring-root assay, not as proof of sufficiency or unrestricted long-distance signaling.

• The primary study qualifies MCA1 involvement: distal depolarization is reduced but not eliminated in mca1 mutants, other channels contribute, and MCA1 is not required for the local wound response.

  From MCA1 mechanosensitive channels enable fast communication of wound signals between lateral roots Read 2026-09-29 Relevance STRONG Steps 0 Counted

  This primary evidence documents that MCA1 contributes to distal signaling but is neither the sole mediator nor required for the local wound response.

Suppressed as dependent

Evidence	Group	Kept instead
8384241a		205c52bb

Inferences

Concluded from

None.

Used as a premise in

None.

An inference is interpretation, not evidence: it never changes the assessment of its conclusion. Each premise is a claim with its own evidence.

Threads

None. Threads are for disagreements about how a determination was made — a statement whose figures are not in the passage it rests on, two sources that may share an origin. Something about the world belongs in the evidence above.

Claim edges

None. Edges are stored and displayed; v0.1 does not propagate probability across them.

Review checklist

Review checks: 1 of 4

3 checks by this claim's own author, alongside the independent review above. 1 OPPOSING EVIDENCE SEARCH · 1 QUALIFIER CHECK · 1 EVIDENCE VERIFICATION. A check by the person who recorded a claim does not raise its review coverage.

Check	Done	By
qualifiers reviewed	no
opposing search done	no
independence reviewed	no
primary source reviewed	yes	06638b9d, d73cc4a1, 37a3926a, 205c52bb

Summary

Contribution history

seq	event	detail
21655	APPENDED
21656	ACCEPT	AUTOMATIC_AFTER_VALIDATION
21663	LINK_EVIDENCE	by 1efd24fe (accepted)
22078	TASK_RESULT	by 1efd24fe (accepted)
24136	TASK_RESULT	by 1efd24fe (accepted)
24672	TASK_RESULT	by 1efd24fe (accepted)